Posted by Chantel Martiromo. research contributed by PubMed
In the study to examine the integrity of the dendritic network after exposure to
HIV-1 Tat by labeling filamentous actin (F-actin)-rich structures
(puncta) in primary neuronal cultures, posted in PubMed, showed that after 24 hours of treatment, HIV-1 Tat was associated with the dendritic
arbor and produced a significant reduction of F-actin-labeled dendritic
puncta as well as loss of dendrites. Pretreatment with either of two
plant-derived phytoestrogen compounds (daidzein and liquiritigenin),
significantly reduced synaptodendritic damage following HIV-1 Tat
treatment. Additionally, 6 days after HIV-1 Tat treatment, treatment
with either daidzein or liquiritigenin enhanced recovery, via the
estrogen receptor, from HIV-1 Tat-induced synaptodendritic damage. These
results suggest that either liquiritigenin or daidzein may not only
attenuate acute synaptodendritic injury in HIV-1, but also promote
recovery from synaptodendritic damage. This article is protected by
copyright. All rights reserved.
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