Posted by Chantel Martiromo, Research article by Kyle J. Norton
During the last stage of the menstrual cycle, normally a layer of
endometriosis lining on the inside of the uterus is expelled, known as
menstruation blood, instead some of the endometriosis tissues grow
somewhere in the body to cause endometriosis. Endometriosis also react
to hormone signals of the monthly menstrual cycle, by building and
breaking up tissues and eliminating them through menstrual period.
The Effects on n Digestive System
1. Constipation or difficult defecation
Defecation is the final act of digestion by which organisms eliminate
waste material from the digestive tract via the anus. Constipation is
caused by endometriosis implants or adhesion thst are attached to organs
of excretion resulting in painful urination, as well as defecation(60)(61).
2. Bloating
Bloating may have several causes, the most common being accumulation of
liquids and intestinal gas. In endometriosis, bloating is caused by
ovarian cysts, endometrial implants, or adhesion in the intestines that
promote collection of fluid within an ovary causing swelling in the
abdominal region and swelling in the digestive system as resulting of
lack of regular ovulation and excessive amounts of androgenic hormones(62)(63).
3. Rectal bleeding
Constipation normally is the main cause of rectal bleeding. In
endometriosis, endometrial implants or adhesion in large intestine
resulting in rectal bleeding as waste passing through. It also causes
rectal bleeding just before and during menstruation as endometrial
bleeding(64)(65).
4. Diarrhea
Sudden bouts of diarrhea may be caused by bacterial infection. In woman
with endometriosis, deficiency of vitamin A and B are common, causing
low level of enzymes that allow food to pass through the intestine
undigested(66).
5. Fluid retention
Fluid retention may be caused by endometrial cysts located other than in
the reproductive system. Endometrial lining bleeding during
menstruation causes blood to have nowhere to escape causing fluid
retention in some parts of the body(67).
6. Anorexia
Anorexia nervosa is a type of eating disorder usually develop in the
teen years and effect over 90% of female, because of excessive food
restriction and irrational fear to become fat due the wrongly influenced
media as attractiveness is equated to thinness. Some researchers suggested that the earliest signs of the disease:
anorexia, dysmenorrhea, menorrhagia, irregular menstrual cycles, or infertility may
be an indication of endometriosis, in animal study(68).
F. Difficult in Urination(10)
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References
(60) Irritable bowel syndrome and chronic
constipation in patients with
endometriosis by Meurs-Szojda MM
1, Mijatovic V, Felt-Bersma RJ, Hompes PG.(
PubMed)
(61)[Transmural rectal
endometriosis as a cause of chronic
constipation. A case report and literature review].[Article in Spanish] by Hernández Magro PM
1, Barrón Vega R, Rivas Larrauri E, Godínez Guerrero MA, Pérez González M, López Patiño S.(
PubMed)
(62)[
Endometriosis of the ileum as cause intestinal obstruction].[Article in Spanish] by Meza Flores JL
1, Ruiz Yaya K, Ramírez Anglas FL, Yabar Berrocal A.(
PubMed)
(63)Abdominal
bloating: an under-recognized
endometriosis symptom by Luscombe GM
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PubMed)
(64)Intestinal
endometriosis as a cause of
rectal bleeding: a case report by Vergelí Rojas JA
1, Pagán Rodríguez L, Santiago Muñoz C, Gutiérrez Rivera S.(
PubMed)
(65) Cyclical
rectal bleeding in colorectal
endometriosis by Levitt MD
1, Hodby KJ, van Merwyk AJ, Glancy RJ.(
PubMed)
(66) Randomized trial of laparoscopically assisted versus open colorectal resection for
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PubMed)
(67)Massive hemothorax due to diaphragmatic
endometriosis after a laparoscopic cystectomy of an ovarian endometrioma in a patient without a history of thoracic
endometriosis by Kyo S
1, Takakura M, Nishida S, Ozaki S, Oda M, Inoue M.(
PubMed)
(68)
endometriosis should be suspected in animals displaying the earliest signs of the disease:
anorexia, dysmenorrhea, menorrhagia, irregular menstrual cycles, or infertility by Green SL
1, Tolwani RJ, Waggie KS, Otto GM. (
PubMed)